Market Cap 90.31M
Revenue (ttm) 266.96M
Net Income (ttm) 28.51M
EPS (ttm) N/A
PE Ratio 0.00
Forward PE N/A
Profit Margin 10.68%
Debt to Equity Ratio -0.20
Volume 1,822,400
Avg Vol 1,708,078
Day's Range N/A - N/A
Shares Out 115.93M
Stochastic %K 30%
Beta 1.01
Analysts Strong Sell
Price Target $5.26

Company Profile

Coherus Oncology, Inc., a biopharmaceutical company, researches, develops, and commercializes immunotherapies to treat cancer in the United States. The company develops UDENYCA, a biosimilar to Neulasta, a long-acting granulocyte-colony stimulating factor; LOQTORZI, a novel next-generation programmed death receptor-1 inhibitor; and Casdozokitug, an investigational recombinant human immunoglobulin isotype (IgG1) monoclonal antibody targeting interleukin IL-27. It also develops CHS-114, an investi...

Industry: Biotechnology
Sector: Healthcare
Phone: 650 649 3530
Address:
333 Twin Dolphin Drive, Suite 600, Redwood City, United States
Jack_Nicholson_Truth
Jack_Nicholson_Truth Apr. 24 at 9:29 PM
$CHRS another scam artist. Tell us more 30 years experience scammer. You have like one post every 6 months and clearly a scam account. Your choices are horrible so stop spreading your fud
0 · Reply
bioinsights
bioinsights Apr. 24 at 5:43 PM
$CHRS Full disclosure I am long 50K shares. Additionally I have 30 years in Biotech development in IO and fair balance is key. The MOA statement is mechanistic positioning, not proven clinical differentiation. Best-case clinical interpretation: Toripalimab may work in a broader patient population (less PD-L1 dependence) and drive somewhat stronger or more durable responses. Most likely reality (based on PD-1 class experience): Differences vs Pembrolizumab are incremental. Clinical outcomes will be driven more by: Tumor type, combination strategy (chemo, IO, targeted agents) and line of therapy. Clinical bottom line. Single-agent reality (class-wide): Most PD-1 inhibitors—including Toripalimab and Pembrolizumab—cluster around ~15–30% ORR in most solid tumors. Higher only in highly immunogenic settings. For toripalimab no clear evidence of meaningfully higher single-agent response rates. Mechanistic advantages (binding, agonism) have not translated into step-function clinical gains.
2 · Reply
Grandgustav
Grandgustav Apr. 24 at 2:38 PM
0 · Reply
cdlawton07
cdlawton07 Apr. 24 at 12:43 PM
$CHRS @bioinsights you shorting CHRS or something? Binding the FG loop can cause the cell to internalize the PD-1 receptor (the cell retracts the receptor from the surface and internalizes it). This is different than Keytruda binding to the receptor to simply block it. "Overall, our study demonstrates that toripalimab is differentiated from pembrolizumab in terms of stronger PD-1 binding, more potent in-vitro T cell activation and lower agonistic potential. These characteristics of toripalimab present it as a next generation PD-1 checkpoint inhibitor with potential for favorable clinical outcomes in treating cancer patients irrespective of their PD-L1 status." https://aacrjournals.org/mct/article/22/12_Supplement/C069/730343/Abstract-C069-Toripalimab-an-anti-PD-1-antibody But go ahead and keep trying to spread your disinformation.
0 · Reply
bioinsights
bioinsights Apr. 24 at 12:23 PM
$CHRS “10-fold more binding” = 10× tighter PD-1 affinity, but in the PD-1 class, it is rarely clinically meaningful on its own because current drugs already operate at or near maximal receptor occupancy.Most PD-1 drugs already achieve >90–95% receptor occupancy at standard doses Once saturated, more affinity ≠ more clinical effect. Where it can matter? Lower dose potential (if occupancy achieved at lower exposure). Competitive differentiation in crowded IO space (more marketing than biology in many cases). Edge cases: low antigen density or high PD-L1 expression tumors
0 · Reply
calibran_de
calibran_de Apr. 23 at 8:16 PM
$CHRS https://www.tipranks.com/news/blurbs/oppenheimer-reaffirms-their-buy-rating-on-coherus-biosciences-chrs-blurbs-news?utm_source=edition.cnn.com&utm_medium=referral
0 · Reply
BigDaddyCapital
BigDaddyCapital Apr. 23 at 7:35 PM
$CHRS whether you like this or not it is partially true. Which is why sp has been like a zombie company
1 · Reply
AlbertoBurgio
AlbertoBurgio Apr. 23 at 6:59 PM
$CHRS thank you for posting, I was waiting for the Samishere pattern to form. Now the signal is on.
1 · Reply
Samishere
Samishere Apr. 23 at 6:20 PM
$CHRS Yep Yep ..and Yep!
1 · Reply
cdlawton07
cdlawton07 Apr. 23 at 5:25 PM
$CHRS What are you on about? No one is saying that CHRS can immediately apply for FDA approval in Loqtorzi indications using the China data without U.S trials. "Small differences in MOA do not matter." You consider Loqtorzi being able to treat patients and produce results that Keytruda is unable to replicate "not mattering"? Won't be paying your posts any mind going forward if thats the case.
0 · Reply
Latest News on CHRS
Coherus Oncology to Participate in Upcoming Investor Conferences

Feb 23, 2026, 9:30 AM EST - 2 months ago

Coherus Oncology to Participate in Upcoming Investor Conferences


Coherus to Participate in Upcoming Investor Conferences

May 1, 2025, 5:04 PM EDT - 1 year ago

Coherus to Participate in Upcoming Investor Conferences


Why Is Coherus BioSciences Stock Trading Higher On Tuesday?

Dec 3, 2024, 9:34 AM EST - 1 year ago

Why Is Coherus BioSciences Stock Trading Higher On Tuesday?


Coherus Completes Divestiture of Ophthalmology Franchise

Mar 4, 2024, 1:00 AM EST - 2 years ago

Coherus Completes Divestiture of Ophthalmology Franchise


Coherus BioSciences Announces New Employment Inducement Grants

Feb 23, 2024, 5:33 PM EST - 2 years ago

Coherus BioSciences Announces New Employment Inducement Grants


Jack_Nicholson_Truth
Jack_Nicholson_Truth Apr. 24 at 9:29 PM
$CHRS another scam artist. Tell us more 30 years experience scammer. You have like one post every 6 months and clearly a scam account. Your choices are horrible so stop spreading your fud
0 · Reply
bioinsights
bioinsights Apr. 24 at 5:43 PM
$CHRS Full disclosure I am long 50K shares. Additionally I have 30 years in Biotech development in IO and fair balance is key. The MOA statement is mechanistic positioning, not proven clinical differentiation. Best-case clinical interpretation: Toripalimab may work in a broader patient population (less PD-L1 dependence) and drive somewhat stronger or more durable responses. Most likely reality (based on PD-1 class experience): Differences vs Pembrolizumab are incremental. Clinical outcomes will be driven more by: Tumor type, combination strategy (chemo, IO, targeted agents) and line of therapy. Clinical bottom line. Single-agent reality (class-wide): Most PD-1 inhibitors—including Toripalimab and Pembrolizumab—cluster around ~15–30% ORR in most solid tumors. Higher only in highly immunogenic settings. For toripalimab no clear evidence of meaningfully higher single-agent response rates. Mechanistic advantages (binding, agonism) have not translated into step-function clinical gains.
2 · Reply
Grandgustav
Grandgustav Apr. 24 at 2:38 PM
0 · Reply
cdlawton07
cdlawton07 Apr. 24 at 12:43 PM
$CHRS @bioinsights you shorting CHRS or something? Binding the FG loop can cause the cell to internalize the PD-1 receptor (the cell retracts the receptor from the surface and internalizes it). This is different than Keytruda binding to the receptor to simply block it. "Overall, our study demonstrates that toripalimab is differentiated from pembrolizumab in terms of stronger PD-1 binding, more potent in-vitro T cell activation and lower agonistic potential. These characteristics of toripalimab present it as a next generation PD-1 checkpoint inhibitor with potential for favorable clinical outcomes in treating cancer patients irrespective of their PD-L1 status." https://aacrjournals.org/mct/article/22/12_Supplement/C069/730343/Abstract-C069-Toripalimab-an-anti-PD-1-antibody But go ahead and keep trying to spread your disinformation.
0 · Reply
bioinsights
bioinsights Apr. 24 at 12:23 PM
$CHRS “10-fold more binding” = 10× tighter PD-1 affinity, but in the PD-1 class, it is rarely clinically meaningful on its own because current drugs already operate at or near maximal receptor occupancy.Most PD-1 drugs already achieve >90–95% receptor occupancy at standard doses Once saturated, more affinity ≠ more clinical effect. Where it can matter? Lower dose potential (if occupancy achieved at lower exposure). Competitive differentiation in crowded IO space (more marketing than biology in many cases). Edge cases: low antigen density or high PD-L1 expression tumors
0 · Reply
calibran_de
calibran_de Apr. 23 at 8:16 PM
$CHRS https://www.tipranks.com/news/blurbs/oppenheimer-reaffirms-their-buy-rating-on-coherus-biosciences-chrs-blurbs-news?utm_source=edition.cnn.com&utm_medium=referral
0 · Reply
BigDaddyCapital
BigDaddyCapital Apr. 23 at 7:35 PM
$CHRS whether you like this or not it is partially true. Which is why sp has been like a zombie company
1 · Reply
AlbertoBurgio
AlbertoBurgio Apr. 23 at 6:59 PM
$CHRS thank you for posting, I was waiting for the Samishere pattern to form. Now the signal is on.
1 · Reply
Samishere
Samishere Apr. 23 at 6:20 PM
$CHRS Yep Yep ..and Yep!
1 · Reply
cdlawton07
cdlawton07 Apr. 23 at 5:25 PM
$CHRS What are you on about? No one is saying that CHRS can immediately apply for FDA approval in Loqtorzi indications using the China data without U.S trials. "Small differences in MOA do not matter." You consider Loqtorzi being able to treat patients and produce results that Keytruda is unable to replicate "not mattering"? Won't be paying your posts any mind going forward if thats the case.
0 · Reply
Samishere
Samishere Apr. 23 at 5:13 PM
$CHRS Where are the uber pump chumps Neofraud & Jackass hiding today? No Data ( as predicted) A bag of Phase 1 maybes And a mediocre Chinese PD-1 that cant be acquired without multiple strings attached ( Chinese strings to boot) Oh yeah, and a scumbag CEO & management GLTA!
1 · Reply
bioinsights
bioinsights Apr. 23 at 5:10 PM
$CHRS Small differences in MOA do not matter. What is important for the CHRS PD1 strategy is future clinical trials given the Tori data in China does not transfer easily to the US due to study design and patient population differences. This is where most China-only programs fail to translate to the US reulatory environment. Our FDA requires the following: Results must be generalizable to U.S. patients. Standard of care must match U.S. practice. Investigators and sites must be credible and inspectable. If not, FDA will require additional U.S. or multi-regional trials.
1 · Reply
whateverhappenedthere
whateverhappenedthere Apr. 23 at 5:09 PM
$CHRS I guess Denny embargoed any good news until after the option price reset 😂 May 28 expect some banger PRs I hope!
3 · Reply
cdlawton07
cdlawton07 Apr. 23 at 2:38 PM
$CHRS If they are mechanistically equivalent; why does Tori outperform Pembro in patients with low PD-L1 status?
0 · Reply
Samishere
Samishere Apr. 23 at 1:49 PM
$CHRS .75 cents here we come! This garbage is a shorts dream. Horrible bullshitting management Constant dilution Phase1 big fat maybes that get retail idiots excited One mediocre Chinese product they don’t even own By the why, Junshi can put the kabash on any acquisition involving Tori,, yea look it up! No ones buying a Chinese rental! .75 cents coming soon
0 · Reply
BigDaddyCapital
BigDaddyCapital Apr. 23 at 1:47 PM
$CHRS people dumping on no data. Big candles to start the day
1 · Reply
simonig1
simonig1 Apr. 23 at 1:00 PM
$CHRS Forecast came up this morning: link: https://www.tipranks.com/stocks/chrs/forecast
0 · Reply
Thomas105
Thomas105 Apr. 23 at 12:47 PM
$CHRS new oppenheimer rating from today
1 · Reply
bioinsights
bioinsights Apr. 23 at 11:41 AM
$CHRS The US FDA will not accept chineses data from studies unles it follows FDA guidence. Having said that there is zero difference between the proudcts from an MOA /biologic activity perspective. Toripalimab and Pembrolizumab share the same MOA: both are IgG4 monoclonal antibodies that bind PD-1 on T cells, blocking PD-L1/PD-L2 interactions and restoring antitumor immune activity. Toripalimab binds a slightly different PD-1 epitope, but this does not meaningfully change function. Both avoid T-cell depletion and act by reactivating exhausted T cells. Overall, they are mechanistically equivalent, with differences driven by clinical positioning rather than biology.
4 · Reply
Samishere
Samishere Apr. 23 at 1:13 AM
$CHRS It’s approved for one indication, Head & Neck,, wtf are talking about
1 · Reply
simonig1
simonig1 Apr. 22 at 7:36 PM
$CHRS You're a good bullshitte artist Sam. What you call that "mediocre Chinese PD-1" is approved in cases where Keytruda is not (does NOT work)!!!
2 · Reply
Samishere
Samishere Apr. 22 at 6:41 PM
$CHRS A good chunk of those 28 are small, single-center studies from China (e.g., "Toripalimab in rare salivary gland tumors" or "Small-scale study in bladder cancer"). These add to the bullshitte 28-count but don't typically impact the FDA's view or the stock's valuation. IE- old news. There’s only two worth mentioning , Phase 1b data involving Tagmokitug (CCR8) or Casdozokitug (IL-27), and well, Phase 1…, meaning YEARS away from anything! This 💩 company is nothing more than a distributer of a mediocre Chinese PD-1! Nothing more !
0 · Reply